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Genetic Archaeology
GENETIC ARCHAEOLOGY // PROFILE

TNF

Tumor Necrosis Factor

CHR 6
6p21.33

Overview

TNF (formerly known as Tumor Necrosis Factor-alpha) is one of the most important signaling proteins (cytokines) of the immune system. It coordinates the body’s inflammatory response to infections and injuries.

📍 Chromosomal Position

6p21.33 (Chromosome 6)

🧬 Gene Category

Immune System

🔬 Inheritance

Complex

📊 Prevalence (rs1800629)

A allele: ~15-20% of the European population

Function & Significance

TNF acts as an “alarm button” for the immune system. When released, it triggers cascades that activate defense cells and initiate inflammatory processes. While this is vital for survival in the short term, permanently elevated production of TNF can lead to chronic inflammation and autoimmune diseases.

⚠️ Chronic Inflammation

Certain genetic variants can cause the body to release TNF faster or in larger quantities, which can increase susceptibility to inflammatory conditions.

🧬 Relevant SNP: rs1800629

This SNP is located in the promoter region (the “control unit”) of the TNF gene and influences how much protein is produced:

rs1800629
6:31575254
Allele 1

G

/

Allele 2

A

Interpretation:
G: The more common variant with “normal” production.
A: Associated with increased binding of transcription factors and higher TNF secretion. Carriers of the A allele may exhibit a stronger inflammatory tendency when stimulated.

📚 Data Sources

  • OMIM: #191160 – Tumor Necrosis Factor; TNF
  • dbSNP: rs1800629
  • ClinVar: Variants with clinical significance in immunology.

Biological Function

TNF is involved in systemic inflammation and plays a key role in the immune response to infections. Overexpression is associated with chronic inflammatory diseases.

Associated Conditions

Rheumatoid Arthritis Crohn's Disease Psoriasis Septic Shock
Molecular Analysis

Analyzed Markers

rs1800629 Risk Factor
Pos: 6:31575254 | Alleles: G/A

TNF-308G>A - A-allele associated with increased TNF production and stronger inflammatory responses.